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Splanchnic vasoconstriction in hyperthermic man - Role of falling blood pressure.Results of a study in which six supine resting subjects, wearing water-perfused suits, had body skin temperature controlled at 35 C for 30 min (control period), then rapidly increased to 40.5 C for 43 to 50 min (heating period) in a two-part experiment. In the first part of the experiment arterial mean pressure (MP) in three men was increased back to, or above control levels at the 30 to 35th min of heating by total occlusion of both legs for 8 to 10 min. Splanchnic blood flow (SBF), which had fallen from 1.4 to 0.9 L/min at occlusion, rose only 0.05 L/min during occlusion. Splanchnic vascular resistance (SVR) rose throughout heating and occlusion. In the second part of the experiment (three men) SBF fell despite a spontaneous rise in MP and aortic pulse pressure prior to leg occlusion. Cardiac output (CO) was measured just before, during and after occlusion. Occlusion raised MP 10 to 15 mm Hg and reduced CO only slightly. It is concluded that falling MP or aortic pulse pressure are not major causes of the splanchnic vasoconstriction in response to heating man.
Document ID
19720031551
Acquisition Source
Legacy CDMS
Document Type
Reprint (Version printed in journal)
Authors
Rowell, L. B.
Profant, G. R.
Wyss, C.
(Washington, University Seattle, Wash., United States)
Detry, J.-M. R.
Date Acquired
August 6, 2013
Publication Date
December 1, 1971
Publication Information
Publication: Journal of Applied Physiology
Volume: 31
Subject Category
Biotechnology
Accession Number
72A15217
Funding Number(s)
CONTRACT_GRANT: NIH-RR-37
CONTRACT_GRANT: NGR-48-002-082
Distribution Limits
Public
Copyright
Other

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