A Modeling Study of Oceanic Response to Daily and Monthly Surface Forcing

The goal of this study is to investigate the effect of high-frequency surface forcing (wind stresses and heat fluxes) on upper-ocean response. We use the reduced-gravity quasi-isopycnal ocean model by Schopf and Loughe (1995) for this study. Two experiments are performed: one with daily and the other with monthly surface forcing. The two experiments are referred to as DD and MM, respectively. The daily surface wind stress is produced from the SSM/I wind data (Atlas et al. 1991) using the drag coefficient of Large and Pond (1982). The surface latent and sensible heat fluxes are estimated using the atmospheric mixed layer model by Seager et al. (1995) with the time-varying air temperature and specific humidity from the NCEP-NCAR reanalysis (Kalnay et al. 1996). The radiation is based on climatological shortwave radiation from the Earth Radiation Budget Experiment (ERBE) [Harrison et al. 1993] and the daily GEWEX SRB data. The ocean model domain is restricted to the Pacific Ocean with realistic land boundaries. At the southern boundary the model temperature and salinity are relaxed to the Levitus (1994) climatology. The time-mean SST distribution from MM is close to the observed SST climatology while the mean SST field from DD is about 1.5 C cooler. To identify the responsible processes, we examined the mean heat budgets and the heat balance during the first year (when the difference developed) in the two experiments. The analysis reveals that this is contributed by two factors. One is the difference in latent heat flux. The other is the difference in mixing processes. To further evaluate the responsible processes, we repeated the DD experiment by reducing the based vertical diffusion from 1e-4 to 0.5e-5. The resultant SST field becomes quite closer to the observed SST field. SST variability from the two experiments is generally similar, but the equatorial SST differences between the two experiments show interannual variations. We are investigating the possible mechanisms responsible for the different responses.